Frumusetea vine din interior cu: Produse pentru ingrijirea pielii
Dati tenului prospetimea tineretii Anti Aging.
Ingrediente esentiale naturale: Revitalizante, energizante de reintinerire faciala - Anti Aging.
Frumusetea vine din interior: O fata frumoasa este intotdeauna si o piele sanatoasa, pentru a pastra aceasta armonie intre frumusete si a te simtii bine, este nevoie de ingrijirea pielii cu: Anti-Aging Skin Care®
Anti-Aging Skin Care Products Frumusetea vine din interior Produse pentru ingrijirea pielii
Anti-Aging Skin Care® Products!
Frumusetea vine din interior cu: Produse pentru
ingrijirea pielii!
1. Rolul de DMAE in cosmetica, un produs natural cosmeceutic
DMAE un produs natural pentru ingrijirea pielii
2. DMAE un produs natural pentru ingrijirea pielii, reda tonusul si elasticitatea pierduta
3. Ceai
Verde un produs natural pentru ingrijirea pielii, reda tonusul si elasticitatea
pierduta prin inhibarea de colagenaza
4. DMAE cu efect antirid involvat in citopatologie
5. Stiinta ingrijirii parului: Terapia cu Vitamina B5 (Acid
pantotenic) si Acid paraaminobenzoic care inchide culoarea firului
de par alb.
O deficienta de: Acid paraaminobenzoic (PABA) poate produce stres, dureri
de cap. iritabilitate si par alb.
Acid paraaminobenzoic (PABA) stimuleaza flora intestinala, productia de Vitamina
K, Acid Folic si Tiamina
LONGEVITATEA este importanta, ne mentinem: Longevivi
6. Extract din Samburi de Struguri are actiune de
colagen-elastina, de reintinerire faciala
- Frumusetea vine din interior prin: Extract din Samburi de Struguri
7. Extract din Samburi de Struguri se leaga de
elastina,
cu influentarea de reducerea ratei a elastazei
8. Extract din Samburi de Struguri procianidine inhiba
razele UV B care reduc imunitatea si sint inplicate in riscul dezvoltarii
cancerului de piele
9. Extract din Samburi de Struguri este un aromataza
inhibitor
10. Extract din Samburi de Struguri procianidine activeaza cresterea
foliculului de par si promoveaza proliferarea epiteliala celulara, important
pentru ingrijirea parului
11. Carnosina un Anti-Ageing antioxidant previne degradarea proteica
(proteoliza), reactii de carbonilare grupa de carbonil (> C=O) se leaga
de moleculele de proteina, rezulta oxidarea proteica
- Frumusetea vine din interior prin: Carnosina
- Carnosina pastreaza integritatea structurii proteinelor
de colagen un produs natural pentru ingrijirea pielii, reda tonusul si elasticitatea
pierduta
Grupa de carbonil: Carnosina un Anti-Ageing antioxidant previne
degradarea proteica, Carnosina cu rol in cosmetica, un produs natural
cosmeceutic
12. Carnosina si grupa de carbonil a proteinei o posibila relatie
13. Oxidarea proteinelor si inbatrinirea
14. Carnosina reduce reactiile de glicozilare (AGEs) de incrucisare
(crosslinking) cu glucoza, Carnosine cu efect benefic reduce oxidarea proteinelor
de origine animala
15. Carnosina un supresor al virstei cu activitate de Longevitatee
reduce oxidarea proteinelor de origine animala care este un factor de risc
pentru alte patologii
16. Carnosina reduce reactiile de glicozilare (AGEs)
de incrucisare (crosslinking) cu glucoza, Carnosine cu efect benefic reduce
oxidarea proteinelor de origine animala
17. Carnosina reduce degradarea proteica (proteoliza)
prin peroxidarea lipidica cauza degradarii proteice, reduce reactiile de
glicozilare (AGEs) de incrucisare (crosslinking) cu glucoza si cu efect protectiv
in rectiile de malonilare
- Glicozilare sint reactii necontrolate
ne-enzimatice ale glucozei cu proteinele
Carnosina inhiba reactiile de glicozilare
(AGEs), de brunificare si reducere a eslasticitatii
tesutului de colagen in procesul de imbatrinire
18. MSM (Metilsulfonilmetan) in cosmetica, un produs natural
cosmeceutic
19. Dimetilaminoetanol(DMAE) cu status psihofiziologic de: ma simt
bine (well beeing), la persoane care sufera de dezechilibru
emotional
20. Longevitate cu: Dimetilaminoetanol (DMAE) si
PABA are efect de Longevitate, prin reducerea petelor de batrinete sau
pigmentiilor varstei - lipofuscina.
Este un produs GERO VITAL H3.
21. Efectul Dimetilaminoetanol (DMAE) la longevitate si conportamentul
in virsta
LONGEVITATEA este importanta, intirzie
procesul de imbatrinire, ne mentinem: Longevivi
- Anti Aging
Metode de Longevitate prin reducerea
petelor de batrinete sau pigmentiilor varstei lipofuscina:
LONGEVITATE - GERO VITAL H3
22. Secretul Longevitatii
23. Inbatrinirea de miocite in cultura datorita stresului oxidativ
si a acumularii de lipofuscina
Inbatrinirea de miocite in cultura datorita stresului oxidativ
si a acumularii de lipofuscina
24. Acumularea de Lipofuscina in cerebral pe masura
inaintarii
virstei
25. DMAE cu potential de reversibilitate a acumularii
de
lipofuscina
26. Efectul de DMAE la pigmentii de lipofuscina in sistemul
nervos
27. DMAE cu efect la pigmentii de lipofuscina din neuronii
senili
28.Eliminarea de lipopigments in cerebral
prin terapii de reintinerire
- Anti Aging
Lipofuscina din neuronii senili
Cresterea influxului nervos cu DMAE un precursor
de Acetilcolina
Neuron
Neurotransmitatorii transmit influxul nervos de la neuron spre
neuriti (ramificati de dendrite si axon)
Sinapsa
Exogeneza la nivelul celular
Acetilcolina ACh - Neurotransmitator
Acetilcolinesteraza hidrolizeaza neurotransmitatorul acetilcolina
in acetat si colina
Efecte la nivelul sinapselor
29. Dimetilaminoetanol (DMAE) in longevitate la soareci
senili diferenta
tinar/in virsta (Hochschild R.)
Dimetilaminoetanol (DMAE) cu efect de
longevitate
Hochschild R. a infiintat un scaner care stabileste virsta biologica
umana
30. Dimetilaminoetanol (DMAE) cu efect la performanta cognitiva la
persoanele in virsta
31. Dimetilaminoetanol un precursor de colina, creste
nivelul in cerebral
a acetilcolinei
32. Dimetilaminoetanol si efectul in metabolismul cerebral
a sintezei de acetilcolina
33. Dimetilaminoetanol
(DMAE) indicat la disfunctia
cerebrala progresiva
34. Dimetilaminoetanol
(DMAE) in tratamentul boli Alzheimer, studiu randomizat,
dublu-orb,
placebo controlat
Creier sanatos
Creier cu
Alzheimer
35. Cercetarea
cresterii nivelului de colina prin Dimetilaminoetanol (DMAE)
la dementa degenerativa progresiva
36. Dimetilaminoetanol
(DMAE) in tratamentul de dementa senila
37. Studiu Neuropsihologic cerebral la persoane senile
in timpul tratamentului cu Dimetilaminoetanol
(DMAE)
38. Dimetilaminoetanol (DMAE) cu efect Anti Aging cu proprietati
antioxidative de reducere a radicalilor liberi
39. Baza teoretica a terapiei cu Procaina in tratamentul
longevitatii.
Molecula de Procaina hidrolizeaza in doua fractiuni: Acid paraaminobenzoic
- PABA si dietilaminoetanol (DEAE).
Farmacocinetica absorbtiei la:
PABA - DMAE - DEAE - GERO VITAL H3
- Sa absorbit foarte bine dupa administrare in primele 240 de minute amindoi
metaboliti: PABA si DMAE
- Persoanele in virsta au datorita tesutului o afinitate de absobtie mai
mare
DMAE si PABA - Acid-paraaminobenzoic o alternativa la
Procaina
Procaina prin transformari carboxilice se fractioneaza in saruri de
etanolamina - Dimetilaminoetanol (DMAE), Dietilaminoetanol (DEAE)
40. Experiment clinic farmacologic cu procaina
Structura chimica similara intre DMAE si DEAE
41. Procaina HCL (GERO VITAL H3) un competitiv inhibitor
de monoaminooxidaza
42. Terapia cu Procaina (GERO VITAL H3): mecanismul
inhibitiei de monoaminooxidaza
43. Procaina hidroclorid (GERO VITAL H3) un inhibitor de monoaminooxidaza:
Implicatia terapia de Geriatrie
44. GERO VITAL H3 cu efect in activitatea
de glucoza 6 fosfat dehidrogenaza
45. GERO VITAL H3: Reviu farmacologic
in tratamentul de declin cognitiv
la
virstnici
Informatii din bibliografia electronica la tema: Nutritie si
sanatate
1. Grossman R. The role of dimethylaminoethanol in cosmetic dermatology. Johnson
and Johnson Consumer Products Worldwide, Skillman, NJ 08558, USA. Am J Clin Dermatol.
2005;6(1):39-47
2. Uhoda I, Faska N, Robert C, Cauwenbergh G, Piérard GE. Split face study
on the cutaneous tensile effect of 2-dimethylaminoethanol (deanol) gel. Unit
of Dermocosmetology, Department of Dermatopathology, University Medical Center
of Liège, CHU Sart Tilman, B-4000 Liège, Belgium. Skin Res Technol.
2002 Aug;8(3):164-7.
3. Madhan
B, Krishnamoorthy G, Rao JR, Nair BU. Role of green tea polyphenols in the inhibition
of collagenolytic activity by collagenase. Central Leather
Research
Institute, Adyar, Chennai 600020, India. Int J Biol Macromol. 2007 Jun 1;41(1):16-22.
Epub 2006 Dec 12.
4. The antiwrinkle effect of topical concentrated 2-dimethylaminoethanol
involves
a vacuolar cytopathology.
5. Zarafonetis, CJ. Darkening of gray hair during para-amino-benzoic acid
therapy. J Invest Dermatol. 1950
Dec;15(6):399-401.
- Zvak, C. The Science of Hair Care, 1986, Marcel Dekker, Inc., New York.
- Brandaleone, H., Maine, E., and Steele, J.M. The effect of calcium pantothenate
and para-aminobenzoic acid on gray hair in man. Am J Medical Science, 1944, 206:
315
6. Masquelier J, et al. Stabilization of collagen by procyanidolic oligomers.
Acta Ther 7: 101–105, 1981.
7. Tixier JM, Godeau G, Robert AM, Hornebeck W. Evidence by in vivo and in
vitro
studies that binding of pycnogenols to elastin affects its rate of degradation
by elastases. Biochem Pharmacol. 1984 Dec 15;33(24):3933-9.
8. Sharma SD, Katiyar SK. Dietary grape-seed proanthocyanidin inhibition of
ultraviolet B-induced immune suppression is associated with induction of IL-12.
Department of Dermatology and Skin Diseases Research Center, University of Alabama
at Birmingham, Birmingham, AL, USA and Birmingham VA Medical Center, Birmingham,
AL 35294, USA.Carcinogenesis. 2006 Jan;27(1):95-102. Epub 2005 Jun 29.
9. Kijima I, Phung S, Hur G, Kwok SL, Chen S. Grape seed extract is an aromatase
inhibitor and a suppressor of aromatase expression. Department of Surgical Research,
Beckman Research Institute of the City of Hope, Duarte, California, USA. Cancer
Res. 2006 Jun 1;66(11):5960-7.
10. Takahashi T, Kamiya T, Hasegawa A, Yokoo Y.Procyanidin
oligomers selectively and intensively promote proliferation of mouse hair epithelial
cells in vitro
and activate hair follicle growth in vivo. Tsukuba Research Laboratories, Kyowa
Hakko Kogyo, Ibaraki, Japan.J Invest Dermatol. 1999 Mar;112(3):310-6.
11. Hipkiss
AR, Brownson C, Carrier MJ. Carnosine, the anti-ageing, anti-oxidant dipeptide,
may react with protein carbonyl groups. Division of Biomolecular
Sciences, GKT School of Biomedical Sciences, King's College London, Guy's Campus,
London
Bridge, London SE1 1UL, UK.
Mech Ageing Dev. 2001 Sep 15;122(13):1431-45.
12. Hipkiss AR. Carnosine and protein carbonyl groups: a possible relationship.
Division of Biomolecular Sciences, GKT School of Biomedical Sciences, King's
College London, London SE1 1UL, UK. Biochemistry (Mosc). 2000 Jul;65(7):771-8.
13. Berlett BS, Stadtman ER. Protein oxidation in aging, disease, and oxidative
stress. J Biol Chem. 1997; 272(33):20313-6.
- Stadtman ER. Protein
oxidation and aging. Science. 1992; 257(5074):1220-4.
14. Hipkiss AR. Glycation, ageing and carnosine: are carnivorous diets beneficial
Centre for Experimental Therapeutics, William Harvey Research Institute, John
Vane Science Centre, Bart's and the London Queen Mary's School of Medicine and
Dentistry, Charterhouse Square, London EC1M 6BQ, UK. Mech Ageing Dev. 2005 Oct;126(10):1034-9.
15. Hipkiss AR. Would carnosine or a carnivorous diet help suppress aging and
associated pathologies Centre for Experimental Therapeutics, William Harvey
Research Institute, Barts' and the London School of Medicine and Dentistry, UK.
Ann N Y Acad Sci. 2006 May;1067:369-74.
16. Hipkiss AR, Michaelis J, Syrris P. Non-enzymatic glycosylation of the dipeptide
L-carnosine, a potential anti-protein-cross-linking agent. FEBS Lett. 1995; 371(1):81-5.
- Burcham PC, Kuhan YT. Diminished susceptibility to proteolysis after protein
modification by the lipid peroxidation product malondialdehyde: inhibitory role
for crosslinked and noncrosslinked adducted proteins. Arch Biochem Biophys. 1997;
340(2):331-7.
- Brownson C, Hipkiss AR. Carnosine reacts with a glycated protein. Division
of Biomolecular Science, GKT School of Biomedical Sciences, King's College London,
Guy's Campus, London Bridge, London,
UK. Free Radic Biol Med. 2000 May 15;28(10):1564-70.
Hipkiss AR, Preston JE, Himswoth DT, Worthington VC, Abbot NJ. Protective effects
of carnosine against malondialdehyde-induced toxicity towards cultured rat brain
endothelial cells. Molecular Biology and Biophysics Group, King's College London,
Strand, UK. Neurosci Lett. 1997 Dec 5;238(3):135-8.
- Hipkiss AR, Worthington VC, Himsworth DT, Herwig W. Protective effects of carnosine
against protein modification mediated by malondialdehyde and hypochlorite. Molecular
Biology and Biophysics Group, King's College London, UK. Biochim Biophys Acta.
1998 Mar 12;1380(1):46-54.
- Bierhaus A, Hofmann MA, Ziegler R, Nawroth PP. AGEs and their interaction with
AGE-receptors in vascular disease and diabetes mellitus. I. The AGE concept.
Department of Medicine I, University of Heidelberg, Germany. Cardiovasc Res.
1998 Mar;37(3):586-600.
17. Burcham PC, Kuhan YT. Diminished susceptibility to proteolysis after protein
modification by the lipid peroxidation product malondialdehyde: inhibitory role
for crosslinked and noncrosslinked adducted proteins. Arch Biochem Biophys. 1997;
340(2):331-7.
- Brownson C, Hipkiss AR. Carnosine reacts with a glycated protein. Division
of Biomolecular Science, GKT School of Biomedical Sciences, King's College London,
Guy's Campus, London Bridge, London,
UK. Free Radic Biol Med. 2000 May 15;28(10):1564-70.
- Hipkiss AR, Michaelis J, Syrris P. Non-enzymatic glycosylation of the dipeptide
L-carnosine, a potential anti-protein-cross-linking
agent. FEBS Lett. 1995; 371(1):81-5.
Hipkiss AR, Preston JE, Himswoth DT, Worthington VC, Abbot NJ. Protective effects
of carnosine against malondialdehyde-induced toxicity towards cultured rat brain
endothelial cells. Molecular Biology and Biophysics Group, King's College London,
Strand, UK. Neurosci Lett. 1997 Dec 5;238(3):135-8.
- Hipkiss AR, Worthington VC, Himsworth DT, Herwig W. Protective effects of carnosine
against protein modification mediated by malondialdehyde and hypochlorite. Molecular
Biology and Biophysics Group, King's College London, UK. Biochim Biophys Acta.
1998 Mar 12;1380(1):46-54.
- Bierhaus A, Hofmann MA, Ziegler R, Nawroth PP. AGEs and their interaction with
AGE-receptors in vascular disease and diabetes mellitus. I. The AGE concept.
Department of Medicine I, University of Heidelberg, Germany. Cardiovasc Res.
1998 Mar;37(3):586-600.
18. Herschler, R. J., "Methylsulfonylmethane in Cosmetics", 1984,
Cardinal Associates, Inc.
19. Dimpfel W, Wedekind W, Keplinger I. Efficacy of dimethylaminoethanol (DMAE)
containing vitamin-mineral drug combination on EEG patterns in the presence
of different emotional states. Pro Science, Private Research Clinic GmbH -
med. Forschung und Entwicklung -, Kurt-Schumacher-Str. 9, D-35440 Linden, Germany.
w.dimpfel@mewicon.at Eur J Med Res. 2003 May 30;8(5):183-91.
20. Stenback, F., J. H. Weisburger
and G. M. Williams (1988). "Effect
of lifetime administration of dimethylaminoethanol on longevity, aging
changes, and cryptogenic neoplasms in C3H mice." Mech Ageing Dev 42(2):
129-38.
- Ivy GO, Kanai S, Ohta M, Smith G, Sato Y, Kobayashi M, Kitani K. Lipofuscin-like
substances accumulate rapidly in brain, retina and internal organs with cysteine
protease inhibition. University of Toronto, Scarborough, Ontario, Canada. Adv
Exp Med Biol. 1989;266:31-45; discussion 45-7.
21. Cherkin A, Exkardt MJ. Effects
of dimethylaminoethanol upon life-span and
behavior of aged Japanese quail. J Gerontol. 1977 Jan;32(1):38-45.
22.Mann, John. Secrets of Life Extension,
Bantam Books, New York, 1982
23. Terman A, Dalen H, Eaton JW, Neuzil J,
Brunk UT. Aging
of cardiac myocytes in culture: oxidative stress, lipofuscin accumulation, and
mitochondrial turnover. Division of Pathology II, Faculty of Health Sciences,
Linkoping University, SE-58185, Linkoping, Sweden. alete@inr.liu.se Ann N Y Acad
Sci. 2004 Jun;1019:70-7.
24. Goyal VK. Lipofuscin
pigment accumulation in human brain during aging. Exp
Gerontol. 1982;17(6):481-7.
- Dan Riga, Sorin Riga, Florin Halalau, and Francisc Schneider; Brain Lipopigment
Accumulation in Normal and Pathological Aging; Ann. N.Y. Acad.
Sci. 1067: 158–163
(2006).
25. Katz ML. Potential
reversibility of lipofuscin accumulation. Mason Eye Institute, University
of Missouri School of Medicine, One Hospital Drive, Columbia, MO 65212, USA.
Arch Gerontol Geriatr. 2002 May-Jun;34(3):311-7.
26. Riga S, Riga D. Effects of centrophenoxine
on the lipofuscin pigments in the nervous system of old rats. Brain Res. 1974
Jun 7;72(2):265-75.
27. K. NANDY & G. H. BOURNE Effect of Centrophenoxine on the Lipofuscin
Pigments in the Neurones of Senile Guinea-pigs Department of Anatomy, Yerkes
Regional
Primate Research Center of Emory University. Nature 210, 313 - 314 (16 April
1966); doi:10.1038/210313
28.Riga S, Riga D, Schneider F, Halalau
F. Processing,
lysis, and elimination
of brain lipopigments in rejuvenation therapies. Department of Stress Research & Prophylaxis,
Al. Obregia Clinical Hospital of Psychiatry, Bucharest, Romania. Ann N Y Acad
Sci. 2006 May;1067:383-7.
29. Hochschild R. Effect of dimethylaminoethanol
on the life span of senile male
A/J mice. Exp Gerontol 1973, 8(4): 185-191.
30. Marsh GR, Linnoila M. The
effects of deanol on cognitive performance and electrophysiology in elderly
humans. Psychopharmacology (Berl). 1979;66(1):99-104.
- Kostopoulos, G.K. & Phillis, J.W. (1975). “The effects of dimethylaminoethanol
(deanol) on cerebral cortical neurons.” Psychopharmacology Communications;
1(3): 339–47.
31. Haubrich D.R., Wang P.F., D.E. Clody
D.E.,Wedecking P.W. Increase in rat brain acetylcholine induced by choline
or deanol. Life Sci 1975, 17: 975-980.
32. Jope R.S., Jenden D.J. Dimethylaminoethanol
(deanol) metabolism in rat brain and its effect on acetylcholine synthesis.
J Pharmacol Exp Ther 1979, 211:472-479.
33. Lewis,
J. A. and B. S. Lewis (1977). "Deanol in minimal brain dysfunction." Dis
Nerv Syst 38(12 Pt 2): 21-4.
34. Fisman M,
Mersky H, Helmes E. Double-blind trial of 2-dimethylaminoethanol
in Alzheimer’s disease. Am J Psychiatry 1981;138:970–2.
35. Russell RW, Jenden DJ, Booth RA,
Lauretz
SD, Rice KM, Roch M. Global
in vivo replacement of choline by N-aminodeanol. Testing a hypothesis about
progressive degenerative dementia: II. Physiological and behavioral effects.
Department
of Pharmacology, School of Medicine, University of California, Los Angeles
90024. Pharmacol Biochem Behav. 1990 Dec;37(4):811-20.
36.Ferris,
S. H., G. Sathananthan, S. Gershon and C. Clark (1977). "Senile dementia:
treatment with deanol." J Am Geriatr Soc 25(6): 241-4.
37. Bonavita,
E. (1986). "[Neuropsychological study of the senile brain during and after
single and combined treatment with deanol and citicoline]." Clin Ter 117(5):
387-98.
38. Nagy I, Floyd RA. Electron
spin resonance spectroscopic demonstration of the hydroxyl free radical scavenger
properties of dimethylaminoethanol in spin trapping experiments confirming
the molecular basis for the biological effects of centrophenoxine. Arch Gerontol
Geriatr. 1984 Dec;3(4):297-310.
39. Aslan, A. Theoretical bases of
procaine therapy (Gerovital H3 and Aslavital) in the prophylaxis of aging.
Romanian Journal of Gerontology and Geriatrics,
1: 1, 5-15, 1980.
40. Stroescu, V. The experimental
and clinical pharmacology of procaine, Gerovital H3 and Aslavital. Romanian
Journal of Gerontology and Geriatrics, No.4, Volume
9, pp. 427-437. 1988.
41. MacFarlane MD. Procaine
HCl (Gerovital H3): a weak, reversible, fully competitive inhibitor of monoamine
oxidase. Fed Proc. 1975 Jan;34(1):108-10.
42. MacFarlane MD, Besbris H.Procaine
(Gerovital H3) therapy: mechanism of inhibition
of monoamine oxidase. J Am Geriatr Soc. 1974 Aug;22(8):365-71.
43. Fuller RW, Roush BW. Procaine
hydrochloride as a monoamine oxidase inhibitor: implications for Geriatric
therapy. J Am Geriatr Soc. 1977 Feb;25(2):90-3.
44. Cruceanu A, Bucsa L. Age
differences in the glucose-6-phosphate dehydrogenase activity of homogenates
from the liver of rats. The effect of gerovital H3 on the glucose-6-phosphate
dehydrogenase activity. Physiologie. 1979 Jan-Mar;16(1):71-5.
45. Reisberg B, Ferris SH, Gershon S. An
overview of pharmacologic treatment of cognitive decline in the aged. Am
J Psychiatry. 1981 May;138(5):593-600.
46. Olsen EJ, Bank L, Jarvik LF. Gerovital-H3:
a clinical trial as an antidepressant.J Gerontol. 1978 Jul;33(4):514-20.
47.
Verzar F. Note on the influence
of procaine, PABA and DEAE on the aging of rats. Basel, 1959, Gerontology
3,6, 350-355.
Parerea din pasajele acestui site nu a fost independent cercetate sau confirmate.